Ohio State offers expert, comprehensive care for metabolic bone and mineral disorders, including:
- Osteoporosis – loss of bone density and bone quality which increases the risk for bone fracture, including conditions associated with steroids and other medications, transplantation, GI disorders, rheumatologic disorders
- Calcium metabolism disorders, such as hypercalcemia (high blood calcium) and hypocalcemia (low blood calcium)
- Phosphorus metabolism disorders – abnormal phosphate levels (we usually see low blood phosphate levels)
- Parathyroid disorders- such as primary and secondary hyperparathyroidism and hypoparathyroidism – These could be reasons for abnormal blood calcium levels
- Pseudohypoparathyroidism – body fails to respond to parathyroid hormone and shows low blood calcium levels
- Fibrous dysplasia of bone – a bone tumor which may be associated with other endocrine problems.
- Osteogenesis imperfecta – also known as brittle bone disease
- Osteomalacia – bone that are “soft” due to lack of mineralization
- Osteopetrosis – abnormally dense bone that's prone to breakage
- Paget’s disease of bone – localized areas of bone with high bone turnover that can lead to misshapen bone
- Renal Osteodystrophy – a variety of bone conditions due caused by failing kidneys
- X-linked hypophosphatemia – congenital condition of low blood phosphate and bowed legs
- Hypophosphatasia – inherited disorder that can cause osteomalacia, bone pain
- Parathyroid cancer– endocrinologists may assist with medications to control high calcium and high parathyroid hormone levels
Why Choose Ohio State
The Department of Endocrinology at The Ohio State University Wexner Medical Center has been successfully evaluating and treating patients with bone and parathyroid-related disorders for decades. Many of our patients are referred to us by primary care and specialty providers due to our outstanding reputation.
Clinical Research
Our Endocrinology team is active in a number of clinical studies involving osteoporosis, hypoparathyroidism, hypophosphatasia, and X-linked hypophosphatasia.